hepg2 cells (Santa Cruz Biotechnology)
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Santa Cruz Biotechnology
hepg2 cells
Hepg2 Cells, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 8 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/atg3/Atg3+siRNA/pm41713198-60-3-13
Average 93 stars, based on 8 article reviews
Hepg2 Cells, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 8 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/atg3/Atg3+siRNA/pm41713198-60-3-13
Average 93 stars, based on 8 article reviews
hepg2 cells - by Bioz Stars,
2026-09
93/100 stars
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Construct:Article Title: A lysosome membrane regeneration pathway depends on TBC1D15 and autophagic lysosomal reformation proteins. Article Snippet: Acute lysosomal membrane damage reduces the cellular population of functional lysosomes.. However, these damaged lysosomes have a remarkable recovery potential independent of lysosomal biogenesis and remain unaffected in cells depleted in TFEB and TFE3.. We combined proximity-labelling-based proteomics, biochemistry and high-resolution microscopy to unravel a lysosomal membrane regeneration pathway that depends on ATG8, the lysosomal membrane protein LIMP2, the RAB7 GTPase-activating protein TBC1D15 and proteins required for autophagic lysosomal reformation, including dynamin-2, kinesin-5B and clathrin. Plasmid Preparation:Article Title: A lysosome membrane regeneration pathway depends on TBC1D15 and autophagic lysosomal reformation proteins. Article Snippet: Acute lysosomal membrane damage reduces the cellular population of functional lysosomes.. However, these damaged lysosomes have a remarkable recovery potential independent of lysosomal biogenesis and remain unaffected in cells depleted in TFEB and TFE3.. We combined proximity-labelling-based proteomics, biochemistry and high-resolution microscopy to unravel a lysosomal membrane regeneration pathway that depends on ATG8, the lysosomal membrane protein LIMP2, the RAB7 GTPase-activating protein TBC1D15 and proteins required for autophagic lysosomal reformation, including dynamin-2, kinesin-5B and clathrin. Blocking Assay:Article Title: PHB2 binds to ERβ to induce the autophagy of porcine ovarian granulosa cells through mTOR phosphorylation. Article Snippet: Autophagy of ovarian granulosa cells is one of the reasons which results in follicular atresia.. PHB2 regulates many fundamental biological processes and is pivotal in the mitophagy of cells; nevertheless, the autophagy in the porcine ovary and how PHB2 regulates the follicular cells are unknown.. Here we report a protein complex that induces autophagy in porcine granulosa cells (PGCs) through the direct interaction of ERb and PHB2. Incubation:Article Title: PHB2 binds to ERβ to induce the autophagy of porcine ovarian granulosa cells through mTOR phosphorylation. Article Snippet: Autophagy of ovarian granulosa cells is one of the reasons which results in follicular atresia.. PHB2 regulates many fundamental biological processes and is pivotal in the mitophagy of cells; nevertheless, the autophagy in the porcine ovary and how PHB2 regulates the follicular cells are unknown.. Here we report a protein complex that induces autophagy in porcine granulosa cells (PGCs) through the direct interaction of ERb and PHB2. Nucleic Acid Electrophoresis:Article Title: Involvement of Autophagy in the Protective Effects of Ginsenoside Rb1 in a Rat Model of Traumatic Brain Injury. Article Snippet: Background and Objectives No treatment modalities have been identified to prevent neuron damage induced by traumatic brain injury (TBI).. The objective of this study was to investigate whether ginsenoside Rb1 (GS-Rb1) could be utilized to exert neuroprotective effects in TBI.. Methods Lateral fluid percussion injury (LFPI) was used to induce an experimental TBI model. Lewis rats were divided into a GS-Rb1 group (5, 10, 20 mg/kg, intraperitoneally injected daily), a sham group, and a vehicle group. Magnetic Beads:Article Title: NBR1 mediates autophagic degradation of IRF3 to negatively regulate type I interferon production. Article Snippet: In the setting of virus infection, autophagy regulates the synthesis of type I interferon (IFN) via multiple mechanisms to prevent adverse overreaction.. Interferon regulatory factor (IRF) 3, the dominant transcriptional factor of type I IFN, can be degraded via autophagy-lysosomal pathway.. However, the exact regulatory mechanism is not yet well elucidated. |